Bruton's tyrosine kinase mutations in 8 Chinese families with X-linked agammaglobulinemia

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"screening of the bruton tyrosine kinase (btk) gene mutations in 13 iranian patients with presumed x-linked agammaglobulinemia "

x-linked agammaglobulinemia (xla) is an immunodeficiency caused by mutations in the bruton tyrosine kinase (btk) gene. in order to identify the mutations in btk gene in iranian patients with antibody deficiency, 13 male patients with an xla phenotype from 11 unrelated families were enrolled as the subjects of investigation for btk mutation analysis using pcr-sscp followed by sequencing. five di...

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BTK, the tyrosine kinase affected in X-linked agammaglobulinemia.

X-linked agammaglobulinemia (XLA) is a heritable immunodeficiency disorder that is caused by a differentiation block leading to almost complete absence of B lymphocytes and plasma cells. The affected protein is a cytoplasmic protein tyrosine kinase, Bruton's agammaglobulinemia tyrosine kinase (Btk). Btk along with Tec, Itk and Bmx belong to a distinct family of protein kinases. These proteins c...

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Bruton tyrosine kinase (BTK) in X-linked agammaglobulinemia (XLA).

X-linked agammaglobulinemia (XLA) is a heritable immunodeficiency disorder that is caused by a differentiation block leading to almost complete absence of B lymphocytes and plasma cells. The affected protein is a cytoplasmic protein tyrosine kinase, Bruton's agammaglobulinemia tyrosine kinase (Btk). Btk along with Tec, Itk, Bmx and Txk belong to a distinct family of protein kinases. These prote...

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X-LINKED aGaMMaGLObULINEMIa: MOLEcULar GENEtIc assEssMENt OF tHE brUtON's tYrOsINE KINasE GENE IN tHrEE FaMILIEs

Received for publication: May 10, 2007. Revised: Nov. 05, 2007. rEZUMat 1 3rd Pediatric Clinic, Victor Babes University of Medicine and Pharmacy, Timisoara, 2 2nd Pediatric Clinic, University of Medicine and Pharmacy, Targu Mures, 3 Children Clinical Hospital Oradea, 4 Medical and Health Science Centre, University of Debrecen, Hungary

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ژورنال

عنوان ژورنال: Human Mutation

سال: 2000

ISSN: 1059-7794,1098-1004

DOI: 10.1002/(sici)1098-1004(200004)15:4<385::aid-humu21>3.0.co;2-d